i see a lot of people asking how to lower estrogen, but it’s often without context on why. what’s the actual clinical threshold (not just a reference range) where high E2 becomes problematic enough to warrant intervention? a number on a lab sheet isn’t always the full picture, and frankly, crashing E2 (especially with aggressive AI use) seems like a much more immediate problem for things like bone density and mood stability.
are we treating a number or actual symptoms here? and what’s the evidence for these ‘estrogen-blocking’ supps beyond in vitro data or n=1 anecdotes? i’m curious what symptoms people are actually trying to resolve vs.
just optimizing a lab value